Discover top-quality products tailored for scientific and medical research. Request a personalized quote today
to enhance your projects.
Species | Mouse |
Cat.No | AM2840 |
Product Category | MicroRNA Agomir/Antagomir |
Size/Quantity | 2 OD, 4 OD, 50 OD (size by request, 1 OD corresponds to 33 ug) |
Shipping Info | Room Temperature |
Storage | -20°C / -80°C |
Product Type | microRNA Agomir/Antagomir |
Label | FAM, CY3, CY5, etc. (optional) |
Component | mmu-miR-21a-3p Agomir and/or Antagomir (Product Form: Dry Powder) |
Key Features | *cover all human, mouse, and rat miRNAs listed in miRBase |
MIRacle™ mmu-miR-21a-3p miRNA Agomir/Antagomir is a chemically modified synthetic oligonucleotide developed to precisely modulate the activity of endogenous microRNAs. The Agomir functions as a miRNA mimic, effectively enhancing the expression and biological activity of mmu-miR-21a-3p. Whereas the Antagomir serves as a sequence-specific antagonist, inhibiting mmu-miR-21a-3p function. Both formulations are optimized for high stability and efficacy in in vitro and in vivo settings. AcceGen provides the MIRacle™ series in multiple specifications (2 OD, 4 OD, 50 OD) with HPLC purification to accommodate a diverse range of experimental requirements. These reagents are ideal for investigating the role of mmu-miR-21a-3p in pathological processes such as atrial fibrosis and abnormal angiogenesis, particularly in the context of diabetic cardiovascular complications.
When you publish your research, please cite our product as “AcceGen Biotech Cat.# XXX-0000”. In return, we’ ll give you a $100 coupon. Simply click here and submit your paper’ s PubMed ID (PMID).
MIRacle™ mmu-miR-21a-3p Agomir and Antagomir offer targeted modulation of miR-21a-3p activity, supporting functional studies in mouse models. These reagents are ideal for exploring miR-21a-3p’s involvement in atrial fibrosis and dysregulated angiogenesis, especially under diabetic cardiovascular conditions. Their stability and efficiency make them perfect for uncovering miRNA-driven mechanisms in cardiac remodeling and vascular pathology, both in vitro and in vivo.