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Primary Cells

Cynomolgus Monkey Small Intestinal Epithelial Cells

  • BSL

    1

  • 202
Cynomolgus Monkey Small Intestinal Epithelial Cells are isolated from normal Cynomolgus Monkey small Intestinal tissue. Cells are tested for expression of markers using antibody, E-cadherin or ZO-1 Rabbit Polyclonal Antibody by immunofluorescence staining.
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Home | Primary Cells | Animal Primary Cells | Monkey primary cells | Cynomolgus Monkey Small Intestinal Epithelial Cells
Species

Monkey
Cat.No

ABC-H0015Y
Quality Control

All cells test negative for mycoplasma, bacteria, yeast, and fungi.
Product Category Primary Cells
Size/Quantity

1 vial
Cell Type

Epithelial
Shipping Info

Dry Ice
Growth Conditions

37 ℃, 5% CO2
Source Organ

Intestine
Disease

Normal
Biosafety Level

1
Storage

Liquid Nitrogen
Product Type

Monkey Primary Cells

Description

Cynomolgus Monkey Small Intestinal Epithelial Cells
Cynomolgus Monkey Small Intestinal Epithelial Cells are primary epithelial cells isolated from the small intestine of healthy Cynomolgus monkeys (Macaca fascicularis), a non-human primate species used in preclinical studies. These cells originate from the mucosal lining and are terminally differentiated, including mucus-secreting goblet cells and absorptive enterocytes. They exhibit classic epithelial morphology, form tight junctions, and grow as a monolayer under standard conditions (37°C, 5% CO₂). The cells are typically at early passage (P1–P3) to ensure functional integrity. They form a selectively permeable barrier regulating nutrient absorption and protecting against pathogens, and are involved in immunomodulation and secretion of antimicrobial peptides.

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Application

  • The application of cynomolgus monkey small intestinal epithelial cells offers significant potential across fields related to intestinal health and regenerative medicine. By utilizing in vitro models of these cells, researchers can investigate the regenerative potential of intestinal stem/progenitor cells and their potential in addressing degenerative conditions such as colitis, inflammatory bowel disease, intestinal infections, and radiation injury. Understanding the factors and pathways involved in the controlled differentiation of stem/progenitor cells into tissue-specific lineages may significantly aid the development of effective clinical protocols. Moreover, studying the shedding of senescent enterocytes and the disturbance of intestinal homeostasis in inflammatory conditions can provide valuable insights into the underlying mechanisms and potential therapeutic interventions.

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