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Transfected Stable Cell Lines

Human EGFR (R108K) BAF3 Cell Line

  • For research use only

Cat No.

ABC-X0050C
Product Type

Overexpression Stable Cell Lines
Cell Type

Lymphocyte
Species

Human
Host Cell

BAF3
Source Organ

Lymphatic
Disease

Normal
Storage

Liquid Nitrogen

The human EGFR (R108K) BAF3 cell line provides a powerful tool for studying EGFR mutation-driven tumorigenesis and targeted therapy applications.

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  • Human EGFR (R108K) BAF3 Cell Line
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Home | Transfected Stable Cell Lines | Overexpression Stable Cell Lines | Human EGFR (R108K) BAF3 Cell Line

Description

Human EGFR (R108K) BAF3 Cell Line is a genetically engineered model derived from selected murine Ba/F3 parental cell line based on customers’ requirement. EGFR (R108K) BAF3 mutant cell line is generated by stable integration of exogenous human EGFR gene harboring the R108K mutation into Ba/F3 host cells using our optimized lentiviral transduction system.
Target
EGFR is a transmembrane tyrosine kinase receptor regulating cell growth, survival, and differentiation through PI3K/AKT and MAPK signaling pathways. The R108K mutation, located in the extracellular domain, has been implicated in ligand-independent receptor dimerization and activation, contributing to oncogenesis. AcceGen provides customized EGFR-mutant models to support research into drug resistance, structure-function studies, and targeted therapy development. Polyclonal or monoclonal is optional based on customers’ research needs.

Species

Human
Cat.No

ABC-X0050C
Product Category

Transfected Stable Cell Lines
Size/Quantity

1 vial
Cell Type

Lymphocyte
Growth Mode

Suspension
Shipping Info

Dry Ice
Growth Conditions

37 °C, 5% CO₂
Source Organ

Lymphatic
Disease

Normal
Biosafety Level

1
Storage

Liquid Nitrogen
Product Type

Overexpression Stable Cell Lines
Host Cell

BAF3
Quality Control

All cells test negative for mycoplasma, bacteria, yeast, and fungi.

Application

  • The EGFR (R108K) BAF3 Cell Line serves as a robust tool for functional analysis of EGFR activation, ligand-independent signaling, and therapeutic inhibition. It facilitates studies on mutant EGFR-driven tumorigenesis and preclinical testing of EGFR-targeted agents.

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High Viability
To succeed in cell culture
Precision and Reliability
To support a consistent result
Customization Options
Tailed to your research

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