Transfected Stable Cell Lines

Xpress™ Human CD39 Over-expressing Cell Line (CHOK1)

  • For research use only

Cat No.

ABC-X0208C

Product Type

Overexpression Stable Cell Lines

Cell Type

Epithelial

Species

Human

Host Cell

CHOK1

Source Organ

Ovary

Disease

Normal

Storage

Liquid Nitrogen

The Xpress™ Human CD39 Overexpression Cell Line (CHOK1) enables research into ATP metabolism, immune suppression, and tumor microenvironment studies.

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Description

The CD39 CHOK1 Overexpression Cell Line is a genetically engineered model derived from CHOK1 cells based on customers’ requirement. CD39 CHOK1 overexpression cell line is generated by stable integration of exogenous human CD39 into CHOK1 host cells using our optimized transduction of lentiviral vectors. Overexpression clone is validated at gene level by qRT-PCR.
Target
CD39 (ENTPD1) is an ectonucleotidase involved in hydrolyzing ATP and ADP to AMP, contributing to immunosuppression in the tumor microenvironment. CD39 plays a key role in adenosine signaling and Treg-mediated immune evasion. AcceGen offers generation of stable overexpression cell lines targeting any gene of your interest. Polyclonal or monoclonal is optional based on customers’ research needs.

Species

Human

Cat.No

ABC-X0208C

Product Category

Transfected Stable Cell Lines

Size/Quantity

1 vial

Cell Type

Epithelial

Growth Mode

Adherent

Shipping Info

Dry Ice

Growth Conditions

37 °C, 5% CO₂

Source Organ

Ovary

Disease

Normal

Biosafety Level

1

Storage

Liquid Nitrogen

Product Type

Overexpression Stable Cell Lines

Host Cell

CHOK1

Quality Control

All cells test negative for mycoplasma, bacteria, yeast, and fungi.

Application

  • The CD39 CHOK1 Overexpression Cell Line supports screening of inhibitors targeting purinergic signaling, helping advance tumor immunotherapy and inflammation resolution studies.

Inquiring Xpress™ Human CD39 Over-expressing Cell Line (CHOK1)

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High Viability
To succeed in cell culture
Precision and Reliability
To support a consistent result
Customization Options
Tailed to your research

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