For research use only
| Cat No. | ABC-X0396C |
| Product Type | Overexpression Stable Cell Lines |
| Cell Type | Epithelial |
| Species | Rat |
| Host Cell | CHOK1 |
| Source Organ | Ovary |
| Disease | Normal |
| Storage | Liquid Nitrogen |
Engineered CHOK1 cell line expressing rat Muc1, suitable for mucin-related studies, tumor immunology, and antibody validation research workflows.
Rat Muc1 CHOK1 Overexpression Cell Line is a genetically engineered model derived from selected Chinese hamster ovary (CHOK1) parental cell line based on customers’ requirement. Rat Muc1 CHOK1 overexpression cell line is generated by stable integration of exogenous rat Muc1 (Mucin 1) into CHOK1 host cells using our optimized transduction of lentiviral vectors. Overexpression clone is validated at gene level by qRT-PCR.
Target
Muc1 is a transmembrane glycoprotein widely expressed on the apical surface of epithelial cells and is involved in cell signaling, adhesion, and immune response. In pathological conditions such as cancer, Muc1 is often overexpressed and aberrantly glycosylated, contributing to tumor progression and immune evasion.
AcceGen offers generation of stable overexpression cell lines targeting any gene of your interest. Polyclonal or monoclonal is optional based on customers’ research needs.
| Species | Rat |
| Cat.No | ABC-X0396C |
| Product Category | Transfected Stable Cell Lines |
| Size/Quantity | 1 vial |
| Cell Type | Epithelial |
| Growth Mode | Adherent |
| Shipping Info | Dry Ice |
| Growth Conditions | 37 °C, 5% CO2 |
| Source Organ | Ovary |
| Disease | Normal |
| Biosafety Level | 1 |
| Storage | Liquid Nitrogen |
| Product Type | Overexpression Stable Cell Lines |
| Host Cell | CHOK1 |
| Quality Control | All cells test negative for mycoplasma, bacteria, yeast, and fungi. |
This cell line serves as a valuable tool for studying the functional impact of Muc1 overexpression in epithelial biology and tumor immunology. It is ideal for use in cancer biomarker discovery, Muc1-targeted therapeutic screening, and elucidation of mucin-related signaling pathways in rat-derived models.