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| Product Code | A673; RMS 1598; RMS1598 |
| Species | Human |
| Cat.No | ABC-TC0034 |
| Product Category | Tumor Cell Lines |
| Size/Quantity | 1 vial |
| Shipping Info | Dry Ice |
| Growth Conditions | 37 ℃, 5% CO2 |
| Source Organ | Muscle |
| Disease | Ewing sarcoma |
| Biosafety Level | 1 |
| Storage | Liquid Nitrogen |
| Product Type | Human Sarcoma Cell Lines |
The A-673 cell line, derived from the muscle tissue of a 15-year-old female patient diagnosed with Ewing’s Sarcoma, displays a distinctive polygonal morphology. What sets A673 cells apart is their remarkable ability to produce several growth factors with oncogenic potential, while also secreting growth-inhibitory factors, creating a finely balanced environment for regulating cellular growth. These cells have demonstrated their capacity for tumorigenesis by inducing tumor formation in immunosuppressed mice. Moreover, the A673 cell line carries a complex karyotype characterized by a specific translocation involving chromosomes 11 and 22, rendering it a valuable asset in cancer research and serving as a model for investigating Ewing’s Sarcoma.
Why choose A-673 from AcceGen?
The A-673 cell line boasts exceptional features, including high viability and quality, maintained in a sterile environment free from bacteria, fungi, and mycoplasma contamination. Identity is confirmed through STR analysis, and the cells are incubated under optimal conditions and cryopreserved by experienced operators, ensuring their reliability and suitability for research applications.
When you publish your research, please cite our product as “AcceGen Biotech Cat.# XXX-0000”. In return, we’ll give you a $100 coupon. Simply click here and submit your paper’s PubMed ID (PMID).
FOR RESEARCH USE ONLY
The A-673 cell line is an invaluable asset in biological science, offering a robust model for exploring the intricate balance between tumor-promoting and tumor-suppressing factors. These cells are instrumental in constructing xenograft tumor models, enabling the study of factors that influence tumor angiogenesis and the development of inhibitors to impede tumor growth, holding significant promise for potential therapeutic advancements in the treatment of Ewing’s Sarcoma.
