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Primary Cells

Human Bladder Stromal Fibroblasts

  • For research use only

Cat No.

ABC-TC3515
Product Type

Bladder Cells
Cell Type

Stromal Fibroblast
Species

Human
Growth Conditions

37 ℃, 5% CO2
Source Organ

Bladder
Disease

Normal
Storage

Liquid Nitrogen

Fibroblasts are mesenchymal cells derived from the embryonic mesoderm.

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  • Human Bladder Stromal Fibroblasts
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Home | Primary Cells | Human Primary Cells | Urinary System Primary Cells | Human Bladder Stromal Fibroblasts

Description

Human Bladder Stromal Fibroblasts are primary cells isolated from the healthy human urinary bladder tissue, cryopreserved to retain their native biological properties. They exhibit spindle-shaped morphology and adherent growth in a monolayer. Functionally, they contribute to normal bladder tissue homeostasis synthesizing and organizing the bladder stroma’s extracellular matrix (ECM). Their abnormalities have been shown to be associated with bladder diseases like interstitial cystitis and fibrosis. These fibroblasts express vimentin. They can be passaged multiple times under proper culture conditions. For optimal performance, repeated freezing and thawing should be avoided during culture. The cells undergo rigorous screening and isolation procedures, and are rigorously tested to ensure they are free of contamination from HIV-1, HBV, HCV, Syphilis, Mycoplasma, Fungi, Yeast, and Bacteria.

Product Code

Human Bladder Stromal Fibroblasts, Bladder Stromal Cells Human, Human Bladder Connective Tissue Fibroblasts, HBSFs, Bladder Fibroblasts Stromal
Species

Human
Cat.No

ABC-TC3515
Product Category

Primary Cells
Size/Quantity

1 vial
Cell Type

Stromal Fibroblast
Growth Mode

Adherent
Shipping Info

Dry Ice
Growth Conditions

37 ℃, 5% CO2
Source Organ

Bladder
Disease

Normal
Biosafety Level

1
Storage

Liquid Nitrogen
Product Type

Bladder Cells
Quality Control

All cells test negative for mycoplasma, bacteria, yeast, and fungi.

Application

  • Human Bladder Stromal Fibroblasts can be used as an in vitro cell model to study the pathogenesis of bladder-associated disorders such as interstitial cystitis and bladder fibrosis. Their preserved extracellular matrix regulatory functions enable investigation of critical pathological mechanisms including abnormal extracellular matrix remodeling (activation of matrix-metalloproteinases and their inhibitors), excessive collagen deposition, and TGF-β-mediated fibrotic signaling pathways. This model provides a better understanding of the normal physiological functions of these fibroblasts and the pathological changes occurring in disease states.

Citation

When you publish your research, please cite our product as “AcceGen Biotech Cat.# XXX-0000”. In return, we’ll give you a $200 coupon. Simply click here and submit your paper’s PubMed ID (PMID).

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