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Human Articular Chondrocytes

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Normal Human Articular Chondrocytes (NHAC-kn) are isolated from tissue taken from the human knee. Articular cartilage is characterized by high wear resistance and poor regenerative qualities. Chondrocytes are the resident cells of cartilage and are responsible for synthesizing a range of collagenous and non-collagenous extracellular matrix macromolecules. These include collagen type II, aggrecan and link […]
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Home | Primary Cells | Human Primary Cells | Motion System Primary Cells | Human Articular Chondrocytes
Product Code

NHAC-kn
Species

Human
Cat.No

ABC-TC3968
Quality Control

All cells test negative for mycoplasma, bacteria, yeast, and fungi.
Product Category Primary Cells
Size/Quantity

1 vial
Cell Type

Chondrocyte
Shipping Info

Dry Ice
Growth Conditions

37 ℃, 5% CO2
Disease

Normal
Biosafety Level

1
Storage

Liquid Nitrogen
Product Type

Cartilage Cells

Description

Human Articular Chondrocytes are derived from human articular cartilage. Following primary culture, these cells are cryopreserved. Human Articular Chondrocytes are involved in cartilage regeneration and repair, as well as specific gene regulation. Studies have shown that Human Articular Chondrocytes are implicated in the pathology of arthritis. These cells are characterized by cell staining for S100 and type II collagen. It is negative for HIV-1, HBV, HCV, mycoplasma, bacteria, yeast, and fungi.

AcceGen Frozen Cells & Cell Lines 1 vial
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When you publish your research, please cite our product as “AcceGen Biotech Cat.# XXX-0000”. In return, we’ll give you a $100 coupon. Simply click here and submit your paper’s PubMed ID (PMID).

Application

  • Human Articular Chondrocytes serve as a control for cellular reprogramming and differentiation studies; enable molecular biology research on cartilage-related receptors, signaling pathways, cytokine activation, and gene regulation; model cartilage degradation in osteoarthritis/rheumatoid arthritis by studying apoptosis, cytotoxicity, and cellular responses; regulate immune responses through T cell proliferation modulation; and investigate chondrogenesis mechanisms involving growth factors and signaling pathways.

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