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Primary Cells

Rat Artery Fibroblasts

  • For research use only

Cat No.

ABC-TC4074
Product Type

Rat Primary Cells
Cell Type

Fibroblast
Species

Rat
Growth Conditions

37 ℃, 5% CO2
Source Organ

Artery
Disease

Normal
Storage

Liquid Nitrogen

Rat Primary Artery Fibroblasts from AcceGen are isolated from tissue of adult Sprague-Dawley Rats.

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  • Rat Artery Fibroblasts
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Home | Primary Cells | Animal Primary Cells | Rat Primary cells | Rat Artery Fibroblasts

Description

Rat Artery Fibroblasts are primary mesenchymal cells isolated from the arterial wall of adult rats. These spindle-shaped, adherent cells synthesize key extracellular matrix (ECM) proteins—including collagen I, fibronectin, and elastin—contributing to arterial structure, compliance, and remodeling during vascular injury or stress. They express fibroblast-specific protein-1 (FSP1/S100A4) and vimentin. Functionally, rat artery fibroblasts respond to angiotensin II by increasing proliferation and ECM deposition via TGF-β–mediated pathways. They also mediate matrix remodeling under cyclic strain, reflecting in vivo arterial mechanical dynamics. Each lot undergoes rigorous screening and isolation procedures and is tested to ensure it is free from contamination by Mycoplasma, Fungi, Yeast, and Bacteria.
Product Code

Rat Artery Fibroblasts, Rat Arterial Fibroblasts, Arterial Fibroblasts, RArFs
Species

Rat
Cat.No

ABC-TC4074
Product Category

Primary Cells
Size/Quantity

1 vial
Cell Type

Fibroblast
Growth Mode

Adherent
Shipping Info

Dry Ice
Growth Conditions

37 ℃, 5% CO2
Source Organ

Artery
Disease

Normal
Biosafety Level

1
Storage

Liquid Nitrogen
Product Type

Rat Primary Cells
Quality Control

All cells test negative for mycoplasma, bacteria, yeast, and fungi.

Application

  • Rat Artery Fibroblasts are extensively used in vascular biology and fibrosis research to investigate the mechanisms of extracellular matrix deposition, vascular stiffness, and fibroblast-to-myofibroblast transition. These cells are ideal for modeling arterial remodeling processes in hypertension, atherosclerosis, and restenosis. They are particularly suitable for studies involving angiotensin II–induced signaling, TGF-β–mediated fibrosis, and cyclic strain–responsive gene regulation. Their ability to simulate in vivo arterial responses makes them valuable tools for therapeutic screening aimed at modulating vascular fibrosis and injury repair.

Citation

When you publish your research, please cite our product as “AcceGen Biotech Cat.# XXX-0000”. In return, we’ll give you a $200 coupon. Simply click here and submit your paper’s PubMed ID (PMID).

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