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Primary Cells

Human Muscle Myoblasts, Diabetes Type I

  • For research use only

Cat No.

ABC-TC3956
Product Type

Skeletal Muscle Cells
Cell Type

Myoblast
Species

Human
Growth Conditions

37 ℃, 5% CO2
Source Organ

Muscle
Disease

Diabetes Type I
Storage

Liquid Nitrogen

Human Muscle Myoblasts–Diabetes Type I support diabetic skeletal muscle biology, differentiation modeling, and insulin sensitivity research.

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  • Human Muscle Myoblasts, Diabetes Type I
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Home | Primary Cells | Human Primary Cells | Motion System Primary Cells | Human Muscle Myoblasts, Diabetes Type I

Description

Human Muscle Myoblasts, Diabetes Type I are primary cells isolated from the skeletal muscle tissue of donors with Type I diabetes. These myogenic precursor cells exhibit characteristic spindle-shaped morphology with adherent growth. Functionally, they retain the capacity to differentiate into myotubes, while displaying altered proliferation and differentiation profiles under diabetic metabolic stress, in association with Type 1 diabetes-related cellular muscle characteristics. The cells express myogenic markers MyoD and desmin. These cells have limited proliferative capacity. Each lot undergoes rigorous screening and isolation procedures, and is rigorously tested to ensure it is free of contamination from HIV-1, HBV, HCV, Syphilis, Mycoplasma, Fungi, Yeast, and Bacteria.​

Product Code

Human Muscle Myoblasts, Diabetes Type I, T1D Muscle Myoblasts, Type I Diabetic Myoblasts, Diabetic Skeletal Myoblasts (T1D)
Species

Human
Cat.No

ABC-TC3956
Product Category

Primary Cells
Size/Quantity

1 vial
Cell Type

Myoblast
Growth Mode

Adherent
Shipping Info

Dry Ice
Growth Conditions

37 ℃, 5% CO2
Source Organ

Muscle
Disease

Diabetes Type I
Storage

Liquid Nitrogen
Product Type

Skeletal Muscle Cells
Quality Control

All cells test negative for mycoplasma, bacteria, yeast, and fungi.

Application

  • Human Muscle Myoblasts from Type I Diabetes patients serve as a critical in vitro model to study diabetes-related myopathy and muscle dysfunction. For instance, analyzing the impaired differentiation, insulin resistance, and oxidative stress response of these cells enables researchers to explore molecular pathways like PI3K/AKT signaling deficits, supporting discovery research and target identification for diabetes-related muscle cellular mechanisms.

Citation

When you publish your research, please cite our product as “AcceGen Biotech Cat.# XXX-0000”. In return, we’ll give you a $200 coupon. Simply click here and submit your paper’s PubMed ID (PMID).

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